Genetic analysis implicates APOE, SNCA and suggests lysosomal dysfunction in the etiology of dementia with Lewy bodies

J. Bras, R. Guerreiro, L. Darwent, L. Parkkinen, O. Ansorge, V. Escott-Price, D.G. Hernandez, M.A. Nalls, L.N. Clark, L.S. Honig, K. Marder, W.M. van der Flier, A.W. Lemstra, P. Scheltens, E. Rogaeva, P. St George-Hyslop, E. Londos, H. Zetterberg, S. Ortega-Cubero, P. PastorT.J. Ferman, N.R. Graff-Radford, O.A. Ross, I. Barber, A. Braae, K. Brown, K. Morgan, W. Maetzler, D. Berg, C. Troakes, S. Al-Sarraj, T. Lashley, Y. Compta, T. Revesz, A. Lees, N. Cairns, G.M. Halliday, D. Mann, S. Pickering-Brown, D.W. Dickson, A. Singleton, J. Hardy

Research output: Contribution to journalArticleAcademicpeer-review

Original languageUndefined/Unknown
Pages (from-to)6139-6146
JournalHuman Molecular Genetics
Volume23
Issue number23
DOIs
Publication statusPublished - 2014

Cite this

Bras, J., Guerreiro, R., Darwent, L., Parkkinen, L., Ansorge, O., Escott-Price, V., ... Hardy, J. (2014). Genetic analysis implicates APOE, SNCA and suggests lysosomal dysfunction in the etiology of dementia with Lewy bodies. Human Molecular Genetics, 23(23), 6139-6146. https://doi.org/10.1093/hmg/ddu334