Roles of PI3K and JAK pathways in viability of retinal ganglion cells after acute elevation of intraocular pressure in rats with different autoimmune backgrounds

Y. Huang, Z. Li, N. Wang, N. van Rooijen, Q. Cui

Research output: Contribution to journalArticleAcademicpeer-review

44 Downloads (Pure)
Original languageUndefined/Unknown
Pages (from-to)78
JournalBMC Neuroscience
Volume9
DOIs
Publication statusPublished - 2008

Cite this

@article{9ff59778836c4908b98608d9750d2acc,
title = "Roles of PI3K and JAK pathways in viability of retinal ganglion cells after acute elevation of intraocular pressure in rats with different autoimmune backgrounds",
author = "Y. Huang and Z. Li and N. Wang and {van Rooijen}, N. and Q. Cui",
year = "2008",
doi = "10.1186/1471-2202-9-78",
language = "Undefined/Unknown",
volume = "9",
pages = "78",
journal = "BMC Neuroscience",
issn = "1471-2202",
publisher = "BioMed Central",

}

Roles of PI3K and JAK pathways in viability of retinal ganglion cells after acute elevation of intraocular pressure in rats with different autoimmune backgrounds. / Huang, Y.; Li, Z.; Wang, N.; van Rooijen, N.; Cui, Q.

In: BMC Neuroscience, Vol. 9, 2008, p. 78.

Research output: Contribution to journalArticleAcademicpeer-review

TY - JOUR

T1 - Roles of PI3K and JAK pathways in viability of retinal ganglion cells after acute elevation of intraocular pressure in rats with different autoimmune backgrounds

AU - Huang, Y.

AU - Li, Z.

AU - Wang, N.

AU - van Rooijen, N.

AU - Cui, Q.

PY - 2008

Y1 - 2008

U2 - 10.1186/1471-2202-9-78

DO - 10.1186/1471-2202-9-78

M3 - Article

VL - 9

SP - 78

JO - BMC Neuroscience

JF - BMC Neuroscience

SN - 1471-2202

ER -