Roles of PI3K and JAK pathways in viability of retinal ganglion cells after acute elevation of intraocular pressure in rats with different autoimmune backgrounds

Y. Huang; Z. Li; N. Wang; N. van Rooijen; Q. Cui

doi:10.1186/1471-2202-9-78

Roles of PI3K and JAK pathways in viability of retinal ganglion cells after acute elevation of intraocular pressure in rats with different autoimmune backgrounds

Y. Huang, Z. Li, N. Wang, N. van Rooijen, Q. Cui

Molecular cell biology and Immunology

Research output: Contribution to journal › Article › Academic › peer-review

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Original language	Undefined/Unknown
Pages (from-to)	78
Journal	BMC Neuroscience
Volume	9
DOIs	https://doi.org/10.1186/1471-2202-9-78
Publication status	Published - 2008

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10.1186/1471-2202-9-78

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title = "Roles of PI3K and JAK pathways in viability of retinal ganglion cells after acute elevation of intraocular pressure in rats with different autoimmune backgrounds",

author = "Y. Huang and Z. Li and N. Wang and {van Rooijen}, N. and Q. Cui",

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publisher = "BioMed Central",

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AU - Huang, Y.

AU - Li, Z.

AU - Wang, N.

AU - van Rooijen, N.

AU - Cui, Q.

PY - 2008

Y1 - 2008

U2 - 10.1186/1471-2202-9-78

DO - 10.1186/1471-2202-9-78

M3 - Article

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VL - 9

SP - 78

JO - BMC Neuroscience

JF - BMC Neuroscience

SN - 1471-2202

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